Frag' FlorenceEvidenz. Klar. Anwendbar.
Uhr 7/8Sources Journal Tree
Easy Demo

Lokaler Crossref-Datenbestand · journal-article

An FGF21-IGFBP1 tumor-liver relay drives musculoskeletal defects in colorectal cancer cachexia

Joshua R. Huot, Urja R. Kamdar, Caleb J. Gammon, Patrick D. Livingston, Fabrizio Pin, Nicholas A. Jamnick, Natalia M. Weinzierl, Chandler S. Callaway, Paul A. Roberson, Alyson L. Essex, Jing Xu, Luis Felipe Cardona Polo, Michael W. Devereaux, Aimee L. Anderson, Colin T. Shearn, Ronald J. Sokol, Schuyler Lee, Bifeng Gao, Meher P. Boorgula, Andrew Goodspeed, Leah J. Novinger, Douglas J. Adams, Andrea Bonetto

Nature Communications · 2026

Vollständiger Abstract

Worum geht es in dieser Arbeit?

Abstract Colorectal cancer (CRC) is frequently accompanied by cachexia, a wasting syndrome characterized by progressive loss of muscle and bone that worsens physical function and survival. Although liver dysfunction is increasingly recognized in cachexia, whether tumor-induced liver signaling contributes directly to musculoskeletal decline remains unclear. Here we show that CRC establishes a tumor-liver endocrine relay in which tumor-derived fibroblast growth factor 21 (FGF21) stimulates hepatic production of the hepatokine insulin-like growth factor binding protein 1 (IGFBP1). IGFBP1 is consistently elevated in patients with CRC and in multiple mouse models, where it promotes osteoclast differentiation, muscle atrophy, and bone loss. Genetic or pharmacological inhibition of IGFBP1 preserves muscle and bone despite persistent tumor burden, whereas deletion of tumor-derived FGF21 suppresses hepatic IGFBP1 production and mitigates musculoskeletal wasting. These findings identify a tumor-liver signaling axis that drives CRC-associated cachexia and highlight the FGF21-IGFBP1 pathway as a therapeutic target to preserve musculoskeletal health.

Bibliografischer Nachweis

Publikationsdaten

Autor:innen
Joshua R. Huot, Urja R. Kamdar, Caleb J. Gammon, Patrick D. Livingston, Fabrizio Pin, Nicholas A. Jamnick, Natalia M. Weinzierl, Chandler S. Callaway, Paul A. Roberson, Alyson L. Essex, Jing Xu, Luis Felipe Cardona Polo, Michael W. Devereaux, Aimee L. Anderson, Colin T. Shearn, Ronald J. Sokol, Schuyler Lee, Bifeng Gao, Meher P. Boorgula, Andrew Goodspeed, Leah J. Novinger, Douglas J. Adams, Andrea Bonetto
Quelle
Nature Communications
Publikation
2026-01-01
Band / Ausgabe
Nicht angegeben
Seiten
Nicht angegeben
ISSN / ISBN
2041-1723
Zitationen
0 laut Crossref
Referenzen
0 hinterlegt

Zitieren

Zitierfähiger Nachweis

Joshua R. Huot, Urja R. Kamdar, Caleb J. Gammon, Patrick D. Livingston, Fabrizio Pin, Nicholas A. Jamnick, Natalia M. Weinzierl, Chandler S. Callaway, Paul A. Roberson, Alyson L. Essex, Jing Xu, Luis Felipe Cardona Polo, Michael W. Devereaux, Aimee L. Anderson, Colin T. Shearn, Ronald J. Sokol, Schuyler Lee, Bifeng Gao, Meher P. Boorgula, Andrew Goodspeed, Leah J. Novinger, Douglas J. Adams, Andrea Bonetto (2026). An FGF21-IGFBP1 tumor-liver relay drives musculoskeletal defects in colorectal cancer cachexia. Nature Communications. https://doi.org/10.1038/s41467-026-77305-6
RIS BibTeX CSL-JSON

Kontext

Themen, Förderung und Nutzung

Lizenzhinweise: Lizenz 1